Predators – The Missing Link in the Explosion of CWD

Chronic Wasting Disease (CWD) is a prion-based disease that is basically human created/human exacerbated.

CWD, like all the other prion-based diseases like Mad Cow, Creutzfeldt-Jakob disease (CJD) are 100% fatal and unlike bacterial and viral disease agents, prions are extremely difficult to neutralize. Normal autoclave methods and incineration are ineffective at rendering prions not infectious.

Lands and objects once contaminated are, for practical purposes, contaminated for the foreseeable future.

State and federal agencies have been grappling with how to contain the spread of the disease with essentially no success. The disease continues to spread rapidly.

Efforts have mainly been based on increased surveillance and increased culling. In spite of these efforts, the spread has only accelerated.

Recently Jim Keen, D.V.M., Ph.D. published a paper titled Big Cats as Nature’s Check Against Disease – A summary of theoretical, empirical, and experimental evidence supporting predator cleansing of CWD in deer and elk herds by mountain lions and wolves.

What the report demonstrates is that large predators are far superior to humans in detecting diseased animals. Predators can detect diseased animals long before obvious symptoms of disease can be observed.

As a result, simply allowing normal predator prey balance i.e.not rendering top level predators functionally extirpated from ecosystems may be the best approach to dealing with the CWD problem we have created.

For those interested in prion diseases and wildlife health, this report is a must read.


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Comments

  1. Bruce Bowen Avatar
    Bruce Bowen

    I was never satisfied that CWD is a prion disease and I think that it is unfortunate that so many agencies simply rolled over and accepted this theory without more rigorous investigation. Hence the disease continues to spread.

    Dr. Frank Bastians years of research was ignored even though he discovered a “new” resistant spiroplasm in brain tissue samples from white tailed deer, sheep and humans. This discovery plus existing knowledge of a known spiroplasm, ( S. mirum strain GT 48), which causes symptoms similar to CWD in lab animals should have led to increased research in this area.

    The presence of Azetidine-2-carboxylic acid (AZE 1) in sugar beet products used in deer supplemental feeds could also be a problem. This chemical is a known protein “buster” and has been shown to damage tissues in small animals.

    The range of investigation should not be constrained or cut off because of one theory.

  2. Anotherview Avatar
    Anotherview

    Ugh – prions cause CWD. Nobody rolled over. The only real reason folks struggle with this linkage is because the implications of prion disease – its environmental persistence, its lethality, its potential for transmission, are hard to accept and address. The science is actually pretty straightforward.

  3. Wayne Tyson Avatar
    Wayne Tyson

    Ok . . . . . The box has been opened. Now for the specifics?

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Author
Jonathan Ratner

Jonathan Ratner has been in the trenches of public lands conservation for nearly 25 years. He started out doing forest carnivore work for the Forest Service, BLM, and the Inter-agency Grizzly Bear Study Team, with some Wilderness Rangering on the Pinedale Ranger District. That work lead him directly to deal with the gross corruption within the federal agencies' range program.

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